Polyclonal hyper-IgE mouse model reveals mechanistic insights into antibody class switch recombination.

نویسندگان

  • Shahram Misaghi
  • Kate Senger
  • Tao Sai
  • Yan Qu
  • Yonglian Sun
  • Kajal Hamidzadeh
  • Allen Nguyen
  • Zhaoyu Jin
  • Meijuan Zhou
  • Donghong Yan
  • Wei Yu Lin
  • Zhonghua Lin
  • Maria N Lorenzo
  • Andrew Sebrell
  • Jiabing Ding
  • Min Xu
  • Patrick Caplazi
  • Cary D Austin
  • Mercedesz Balazs
  • Merone Roose-Girma
  • Laura DeForge
  • Søren Warming
  • Wyne P Lee
  • Vishva M Dixit
  • Ali A Zarrin
چکیده

Preceding antibody constant regions are switch (S) regions varying in length and repeat density that are targets of activation-induced cytidine deaminase. We asked how participating S regions influence each other to orchestrate rearrangements at the IgH locus by engineering mice in which the weakest S region, Sε, is replaced with prominent recombination hotspot Sμ. These mice produce copious polyclonal IgE upon challenge, providing a platform to study IgE biology and therapeutic interventions. The insertion enhances ε germ-line transcript levels, shows a preference for direct vs. sequential switching, and reduces intraswitch recombination events at native Sμ. These results suggest that the sufficiency of Sμ to mediate IgH rearrangements may be influenced by context-dependent cues.

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عنوان ژورنال:
  • Proceedings of the National Academy of Sciences of the United States of America

دوره 110 39  شماره 

صفحات  -

تاریخ انتشار 2013